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Viread-tenvir

Gilead Science, INC

Viread-tenvir


"Viread" contains the Drug / Molecule “Tenofovir Disoproxil”. Viread belongs to a class of antiviral medications called reverse transcriptase inhibitors by competing with the natural substrate, deoxyadenosine 5'-triphosphate and by DNA chain termination.

“Tenvir” is generic version of “Viread” and both contains same  Drug / Molecule “Tenofovir Disoproxil”.


Indication

Viread is used in combination with other antiviral medications in patients with acquired immunodeficiency syndrome (AIDS). Viread is also used for the treatment of chronic hepatitis B infection.

Note

SIDE EFFECTS :

  • Common side effects include nausea, rash, diarrhea, headache, pain, depression and weakness.
  • Severe side effects include high blood lactate and an enlarged liver.
  • Long term use of Viread is associated with nephrotoxicity and bone loss.

INTERACTIONS :

  • Tenofovir interacts with Didnosine and HIV-1 protease inhibitors. Tenofovir increases Didanosine concentrations and can result in adverse effects such as pancreatitis and neuropathy.
  • Tenofovir also interacts with HIV-1 protease inhibitors such as Atazanavir, by decreasing Atazanavir concentrations while increasing Tenofovir concentrations.

OVERDOSE :

  • Do not use more than prescribed dose.
  • If you suspect you may have overdosed, call your healthcare provider.
  • Do not take extra dose to make up for the missed dose.

 

Precaution

  • Viread is a prescription drug and should be used under proper medical guidance and advice.
  • Caution to be taken if patient suffering from renal impairment, hepatomegaly or at other liver disease.

 

Updated On Oct 30, 2019 by Admin

Strength

300 mg

Packing

1 X 30 Tablets (Plastic Container)

Storage

Store at Temperature between 15°C and 30°C.

Viread-tenvir also available as


Fast moving

CIPLA LTD. (INDIA)

Tenvir


“Tenvir” contains the Drug / Molecule called “Tenofovir Disoproxil”.

Tenvir belongs to a class of antiviral medications called reverse transcriptase inhibitors by competing with the natural substrate, deoxyadenosine 5'-triphosphate, and by DNA chain termination.





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